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Useful Vitamins for Diabetes Treatment

The classic diabetes therapy focuses mainly on drugs: anti-diabetic agents and insulin therapy. I think this is a big mistake can be the vital nutrients (especially vitamins) and trace the blood glucose levels are greatly improved.

Vitamin E in diabetes
With this vitamin, you reduce your risk of forming a blood clot. In addition, it reduces the formation of LDL-cholesterol, which can lead to atherosclerosis. Vitamin E is contained in 40 g of walnuts, 19 g of sunflower oil or 200 g of black roots – with these sets you meet your daily requirement of 12 mg of vitamin E.

Vitamin B for diabetic
The vitamins of the B-series provide the nervous system with energy. But even among diabetics, the B-vitamins are lost in large quantities in the urine. Are important in diabetes, vitamins B1 (vitamin B1 in diabetes), B6 and B12. These quantities can be found for example in 240 g of brown rice, 400 grams of bananas, 300 g 200 g avocado Brie, 100g goat’s cheese, 200 ml buttermilk 200 g chanterelles or 400 g of peas.

Folic acid in diabetes
Also, the folic acid is actually a B vitamin. It is absent, this leads to increased homocysteine levels, as many as 30% of all diabetics are affected. This increases the risk of vascular diseases is rising even further, and suffer with it the risk of heart attack or a stroke. You need pg per day 160th Include this amount in 300 g spinach, 300 g of Chinese cabbage, 200 g or 200 g fennel beetroot.

Zinc in diabetes
Even the trace element zinc is increased in diabetics excreted in the urine. Lack of zinc, the glucose-impaired recording continues. Furthermore, there is disruption in wound healing, as well as a general susceptibility to infections.

You need a day 10 to 20 mg of zinc. This amount of zinc can be found in 500 g of whole wheat bread, 100 g wheat germ, 190 g of dried mushrooms or 230 g lean hard cheese. Make a note of these foods than those who are zinc-containing. You should always make time to be back on your diet. However, you can cover your zinc requirement as diabetes through diet alone. I tell my patients with diabetes also eat a respiratory tract infections.

Magnesium in diabetes
Magnesium is needed for all processes, which extract energy. Laboratory values, however, show that half of diabetics have too much low magnesium levels. It is this deficiency can be dangerous. Too low magnesium levels reduces the effect of insulin. Too little magnesium is also partly responsible for damage to the retina. You need 300 to 600 mg per day of magnesium.

Include this amount in 200 g of spinach, 3 bananas, 100 g of sesame seeds, or 100 g of sunflower seeds. Unfortunately, the determination of magnesium in the blood no cash value amount and is therefore not intended for the vast majority of patients.

Taurine in diabetes
Taurine is an amino acid that you take on protein products themselves. Taurine reduces the clumping of platelets, and thereby protects against the formation of blood clots – and thus as a prior heart attack and stroke, diabetes. It is therefore possible to feed with high-quality protein in order to provide themselves with adequate taurine to. Suitable protein sources are common, for example, potatoes with cottage cheese or lentils, legumes.

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Urine Investigation for Diabetes (Sugar Illness)

The urine analysis investigation (urine investigation test) is a very simple test you can perform well themselves. For this you need to do a test strip, the cost you can buy in any pharmacy with cost of 15 pieces for about 20 €.

With this urine investigation test, you can determine your urine sugar completely simply. Test your blood sugar regularly, for example, the morning after rising. Urine glucose (urine sugar) frequently found here, shows this clearly indicates a diabetes (diabetes mellitus). Background: Your kidney begins to excrete sugar in the urine when your blood sugar mg to 150g 180 / dl is.

This test is meaningful only in that one direction. You can not automatically assume that you do not suffer from diabetes, if you notice any of these Urine glucose test strips. If the diabetes has affected your kidneys, then the so-called renal threshold, the point is, is excreted in the urine sugar to be increased. Even at very high blood glucose levels, then a divorce is no more sugar.

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Diabetes Cirrhosis Treatment to Prevent Liver Damage

The treatment of diabetes cirrhosis treatment include : prevent further damage to the liver, treatment of complications of cirrhosis, early detection and prevention of liver cancer, and liver transplantation.

And I will explain more detail the diabetes cirrhosis treatment steps, starting from how to prevent further damage to the liver.

• Eat a balanced diet and a daily multivitamin. PBC patients with impaired absorption of fat soluble vitamins may need supplements of vitamins D and K.
• Avoid drugs (including alcohol) that causes liver damage. All patients with cirrhosis should avoid alcohol. Most patients with cirrhosis induced by alcohol experience an improvement in liver function with abstinence from alcohol. Even patients with hepatitis B and C can significantly reduce liver damage and slow the progression to cirrhosis with alcohol withdrawal.
• Avoid anti-inflammatory drugs (NSAIDs, eg ibuprofen). Patients with cirrhosis may experience a worsening of liver and kidney with NSAIDs.
• Reducing hepatitis B and hepatitis C using anti-viral drugs. All patients with cirrhosis from chronic viral hepatitis are candidates for drug therapy. Some patients may experience a severe deterioration of liver function and / or intolerable side effects during treatment. Therefore, decisions to treat viral hepatitis should be individualized after consultation with physicians experienced in treating liver diseases (hepatologists).
• Removal of blood from patients with hemochromatosis to reduce iron levels and prevent further damage to the liver. In Wilson’s disease, medications can be used to increase copper excretion in the urine to reduce levels of copper in the body and prevent further damage to the liver.
• Remove the immune system with drugs such as prednisone and azathioprine (Imuran) to reduce inflammation of the liver in autoimmune hepatitis.
• Treat patients with primary biliary cirrhosis with a preparation of bile acid, ursodeoxycholic acid (UDCA), also called ursodiol (Actigall). The results of an analysis that combines results from several clinical trials have shown that UDCA improved survival in patients with PBC during 4 years of treatment. The development of portal hypertension has also been reduced by UDCA. Importantly, despite evident benefits, UDCA treatment delays progression and above all, not a cure for PBC. Other drugs such as colchicine and methotrexate may also have benefits for subgroups of patients with PBC.
• Patients with cirrhosis of immunizing against infection with hepatitis A and B to prevent a serious deterioration of liver function. There is currently no vaccine available for immunization against hepatitis C.

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The Most Frequent Cirrhosis Causes

There are some causes of cirrhosis, such as alcohol, non-alcoholic fatty liver disease (NAFLD), cryptogenic cirrhosis, chronic viral hepatitis, Inherited disorders, primary biliary cirrhosis, primary sclerosing cholangitis, autoimmune hepatitis, biliary atresia, and cardiac chronic heart failure as cirrhosis causes problems.

• Alcohol cause hepatic cirrhosis

Alcohol is a very common cause of cirrhosis, especially in the western world. The development of cirrhosis depends on the amount and regularity of alcohol consumption. Chronic high levels of alcohol consumption for liver cell damage. Thirty percent of people drinking daily for at least eight to sixteen ounces of hard liquor or the equivalent of fifteen or more years will develop cirrhosis. Alcohol causes a range of diseases of the liver to be simple and uncomplicated fatty liver (steatosis), to more severe fatty liver with inflammation (nonalcoholic steatohepatitis or hepatitis), cirrhosis.

• Non-alcoholic Fatty Liver Disease (NAFLD)

NAFLD, better known as Non-Alcoholic Fatty Liver Disease refers to a broad spectrum of liver diseases such as alcoholic liver disease, ranging from simple steatosis to non-alcoholic steato-hepatitis (NASH) to cirrhosis. All stages of NAFLD have in common the accumulation of fat in liver cells. The term is used because NAFLD nonalcoholic occurs in people who do not consume excessive amounts of alcohol, however, in many respects, the microscopic image of non-alcoholic fatty liver is similar to what can be seen in liver disease caused by excessive alcohol consumption. Is NAFLD associated with a condition called insulin resistance, which in turn is associated with metabolic syndrome and type 2 diabetes mellitus. Obesity is a major cause of insulin resistance, metabolic syndrome, type 2 diabetes. NAFLD is the most common liver disease in the United States and is responsible for 24% of all liver diseases. In fact, the number of livers that are transplanted from non-alcoholic fatty liver, cirrhosis is related to the increase. The public health administrators that the current epidemic of obesity dramatically increases the development of nonalcoholic fatty liver and cirrhosis in the population.

• Cryptogenic cirrhosis (cirrhosis due to unidentified causes)

Cirrhosis due to unidentified causes, better known as Cryptogenic-Cirrhosis is a common reason for liver transplantation. It’s called cryptogenic cirrhosis because for years doctors have been unable to explain why a proportion of patients with cirrhosis developed. Doctors now believe that cryptogenic cirrhosis due to NASH (nonalcoholic steatohepatitis) caused by long-standing obesity, type 2 diabetes and insulin resistance. The fat in the liver of patients with NASH is estimated to disappear with the onset of cirrhosis, and this makes it difficult for physicians to establish the link between NASH and cryptogenic cirrhosis long. An important clue leading to NASH cryptogenic cirrhosis is the discovery of a large number of Nash in the new liver of patients undergoing liver transplantation in cryptogenic cirrhosis. Finally, a French study suggests that patients with NASH have a similar risk of developing cirrhosis patients with long-term infection with hepatitis C. (See below). However, it is expected that progression to cirrhosis from NASH to be slow and the diagnosis of cirrhosis is usually performed in patients in the sixties.

• Chronic Viral Hepatitis

Viral hepatitis chronic is a condition where chronic hepatitis B or chronic hepatitis C affects the liver for years. Most patients with viral hepatitis develop chronic hepatitis and cirrhosis. For example, most patients infected with hepatitis A recover completely within a few weeks without developing chronic infection. However, some patients infected with hepatitis B and most patients infected with hepatitis C develop chronic hepatitis, which in turn leads to progressive liver disease leading to cirrhosis and sometimes liver cancer.

• Inherited (genetic) disorders

Inherited genetic disorders that cause the accumulation of toxic substances in the liver, leading to tissue damage and cirrhosis. Examples include the abnormal accumulation of iron (hemochromatosis) or copper (Wilson’s disease). In hemochromatosis, patients inherit a tendency to absorb too much iron from food. Over time, iron accumulation in various organs in the body causes cirrhosis, arthritis, heart muscle damage leading to heart failure, and testicular dysfunction causing loss of sexual appetite. Treatment aims to prevent organ damage from the removal of iron in the body by bleeding (blood removal). In Wilson’s disease, is an inherited abnormality in one of the proteins that control copper in the body. Over time, copper accumulates in the liver, eyes and brain. Cirrhosis, tremor, psychiatric disorders and other neurological disorders occur if the condition is not treated quickly. Treatment with oral medication that increases the amount of copper from the body in urine.

• Primary biliary cirrhosis (PBC)

PBC, better known as Primary Biliary Cirrhosis is a liver disease caused by abnormal immune system that is predominantly female. Abnormal immunity in PBC causes chronic inflammation and destruction of small bile ducts in the liver. The bile ducts are the passages in the liver, bile travels to the intestine. Bile is a liquid produced by the liver that contains substances necessary for digestion and absorption of fat in the intestine, and other compounds that are waste products such as bilirubin, a pigment. (Bilirubin is produced by the breakdown of hemoglobin in red blood cells.). Along with the gallbladder, bile ducts form the biliary tract. In the PBC, the destruction of small bile duct blocks the normal flow of bile into the intestine. As the inflammation continues to destroy more of the bile ducts, also extends to destroy liver cells nearby. That the destruction of hepatocytes of products, the scar tissue (fibrosis) forms and propagates in the areas of destruction. The combined effects of ongoing inflammation, scarring, and the toxic effects of waste accumulation leads to cirrhosis.

• Primary sclerosing cholangitis (PSC)

PSC, better known as Primary Sclerosing Cholangitis is a rare disease that occurs frequently in patients with ulcerative colitis. In the PSC, the large bile ducts outside the liver become inflamed, narrowed and blocked. Outflow obstruction of the bile duct in biliary tract infections and jaundice, and eventually causes cirrhosis. In some patients, bile duct injury (usually after surgery) can also cause obstruction and cirrhosis of the liver.

• Autoimmune hepatitis

Autoimmune hepatitis is a liver disease caused by abnormal immune system that occurs most often in women. The abnormal immune activity in autoimmune hepatitis resulting in inflammation and progressive destruction of liver cells (hepatocytes), which eventually leads to cirrhosis.

• Babies can be born without bile ducts (biliary atresia)

Biliary atresia, also known as infant can be born without bile ducts and eventually develop cirrhosis. Other babies are born without enzyme vital for control of sugar that leads to the accumulation of sugars and cirrhosis. On rare occasions, the absence of a specific enzyme can cause cirrhosis and scarring of the lungs (alpha-1 antitrypsin deficiency).

• Other less common causes of cirrhosis include unusual reactions to certain drugs and exposure to toxins, and chronic heart failure (cardiac cirrhosis)

In some parts of the world (especially North Africa), infection of the liver parasites (schistosomiasis) is the most common cause of liver disease and cirrhosis.

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Diabetic Nephropathy and Kidney Disease

Diabetic nephropathy, some people also known as Kimmelstiel-Wilson syndrome and intercapillary glomerulonephritis (nephropatia diabetica) is a progressive disease of the kidneys as a result of a long, not optimally controlled diabetes mellitus – in which poorly treated – the kidney function is more limiting up to dialysis, kidney failure. Around 30% of people with diabetes type 1 and diabetes type 2 occurs approximately 10 years after onset of diabetic nephropathy (nephropathy diabetes), and men are statistically more frequently affected than women, including low birth weight acts as a favorable factor.

The relevant factors in addition to a hereditary predisposition and the long-term quality of glycemic control. Due to the long-standing elevated blood sugar deposits from changes in the renal corpuscles and scarring caused by the natural filtering function of the kidneys fail gradually. The arteries of the renal vessels calcify increasingly, there is an increase in pressure within the renal corpuscle and a general rise in blood pressure.

The onset of the illness passes unnoticed by the patient, when the nephropathy is broken and is not treated, but it usually leads within 2.5 years for chronic renal failure. Only the symptoms of advanced renal injury in the form of foamy urine (due to the increased protein secretion), edema and weight gain or incipient renal failure with itching, exhaustion, headache, anemia, nausea and vomiting are more noticeable. In the laboratory, the excretion of albumin is increased.

The simultaneous determination of albumin and creatinine in urine is the albumin-creatinine ratio a clear indication of diabetic nephropathy :

The value of 30-300 mg / g is known as microalbuminuria (likelihood of nephropathy after 10 years, diabetes mellitus type 1) very high; more than 300 mg / g is called macroalbuminuria (very high probability) even after a shorter diabetes.

If 2 of 3 samples reach these values, it must be of the diabetic nephropathy. At the risk of renal failure is due to the burden of blood vessels in hypertension, the risk of cardiovascular complications increases significantly. Smoking, higher amounts of protein from the diet, elevated blood lipids and obesity potentiate the risk of a heart attack, stroke, thrombosis, or diabetic complications.

Blood sugar must be terminated permanently optimal, lowered blood pressure and so must be the blood fat levels reduced to well below 100. The goal is to reduce protein excretion to less than 0.5 – 1 g per day.

Regular blood and urine tests to the corresponding values of renal function are mandatory. As standard medications are ACE inhibitors or AT1 antagonists, antihypertensive drugs can stop the disease prophylactically.

A well adjusted blood sugar levels, consistent weight control, regular medical checkups and a generally healthy lifestyle (exercise, protein and fats in moderation, do not smoke) to help reduce the risk of diabetic nephropathy, and significantly delay the progression of existing disease.

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