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Usually Signs of Hepatitis A Symptoms

The Hepatitis A symptoms and signs usually appear from 15 to 50 days (2 to 7 weeks) after exposure to hepatitis A virus (HAV), and the average time HAV symptoms appears is 4 weeks. The hepatitis A symptoms and signs are usually mild and can not be observed in children under 6 years. Among older children and adults, early symptoms are similar to those of a stomach virus.

Possible hepatitis A symptoms and signs include:
- Extreme fatigue (tiredness).
- Fever.
- Sore muscles.
- Headaches.
- Pain in the right side of the abdomen below the ribs (when the liver is located).
- Nausea.
- Loss of appetite and weight loss.
- Yellowing of the skin and whites of the eyes (jaundice), sometimes accompanied by dark urine and clay-colored (whitish) stools. Jaundice is less common in children and young adults.

About 15% of people with signs of hepatitis A have symptoms of infection with HAV that performance or the last 6 to 9 months after the first infection occurred.1 The infection of hepatitis A is usually more severe than the first time, but the Arthritis can occur with it. After the infection goes away, the person usually returns to normal health. Rarely, cholestatic hepatitis occurs, which can cause itching and can last throughout the infection.

In general, when signs of hepatitis A symptoms occur, the amount of virus is shed in the feces is in decline. You can still spread the virus, but is less likely to do so after symptoms appear. The symptoms of hepatitis A signs usually last less than 2 months. In other countries, hepatitis A also called as l’hépatite A, la hepatitis A, epatite A, WZW typu A, hepatitt A.

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Risk Factors Hepatitis A Symptoms Increase

People who practice certain jobs or certain behaviors are more likely to get hepatitis A. If you are a member of a group at high risk, should receive the vaccine against hepatitis A.

Risk factors for hepatitis A, include:
- Eating food that was prepared by someone who is infected with hepatitis A and poor hygiene.
- Consuming raw or undercooked shellfish (like oysters or clams).
- Eating raw foods (such as unpeeled fruits or vegetables) and drinking tap water or well water while traveling to countries where hepatitis A is common.
- Living in a community where hepatitis A is common and outbreaks occur (largely a risk factor for young children).
- Living in a house with someone who has hepatitis A.

Lifestyle factors that increase the risk of hepatitis A include:
- Travel to countries where hepatitis A is common.
- Be a man having sex with men.

What are the hepatitis A symptoms?

After being exposed to the virus, it may take 2 to 7 weeks before seeing a sign for him. Hepatitis A symptoms usually last about 2 months, usually common symptoms of Hepatitis A are:
- Feeling very tired
- Feeling sick in the stomach
- Not feeling hungry
- Losing weight without effort
- Pain in the right side of the abdomen under the ribs (if your liver)
- Fever
- Aching muscles

Older persons with hepatitis A may get yellow skin (jaundice) and dark urine and feces bunting. All forms of hepatitis have similar symptoms. Only a blood test can determine if you have hepatitis A or another form of the disease.

Call your hepatitis A doctor immediately if :
- You have signs of hepatitis A.
- A person you live with hepatitis A.
- Did you eat at a restaurant that had a virus outbreak.
- Your child goes to kindergarten where hepatitis A was reported.

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Signs and Symptoms of Hepatic Cirrhosis Complications

Patients with cirrhosis may have little or no liver disease symptoms and liver disease. Some cirrhosis symptoms may be nonspecific, i.e. not suggest that the liver is the cause. Among the most common symptoms and signs of cirrhosis, it’s include : itching, fatigue, loss of appetite, weakness, jaundice (yellowing of the skin) due to accumulation of bilirubin in the blood, and easy bruising of the decreased production of blood coagulation by the diseased liver.

Some cirrhosis patients also develop signs and symptoms of cirrhosis complications. The complications of cirrhosis such as edema and ascites, Spontaneous bacterial peritonitis (SBP), Bleeding esophageal varices described here, and Hepatic encephalopathy, Hepatorenal syndrome, Hepatopulmonaire Syndrome, Hypersplenism and Liver cancer (hepatocellular carcinoma) will be explained in part 2.

Edema and ascites

As liver cirrhosis is severe, the signals are sent to the kidneys retain salt and water in the body. The excess salt and water is first stored in the tissue under the skin of the ankles and legs due to gravity standing or sitting. This fluid buildup is called swelling or edema marks. (Fovea refers to the fact that the pressure of a finger firmly against the ankle or leg with edema causes bleeding in the skin that persists for some time after pressure release. In fact, any pressure, as the elastic of a sock, can be enough to cause pitting.) Swelling is often worse at the end of the day, after standing or sitting and may lower overnight due to the loss the effects of gravity on the position supine. As cirrhosis worsens and more salt and water is conserved, the fluid can also accumulate in the abdominal cavity between the abdominal wall and abdominal organs. This accumulation of fluid (called ascites) causes abdominal bloating, abdominal discomfort, and weight gain.

Spontaneous bacterial peritonitis (SBP)

Of fluid in the abdominal cavity (ascites) is the ideal place for bacteria to grow. Normally, the abdominal cavity contains a very small amount of liquid that is able to resist infection well, and bacteria that enter the abdomen (usually the intestine) are killed or find their place in the door and the liver vein, which killed. In cirrhosis, fluid accumulates in the abdomen can not normally resist infection. In addition, more bacteria find their way from the intestine into the ascites. Therefore, the infection within the abdomen and ascites, known as spontaneous bacterial peritonitis or SBP, is likely to happen. SBP is a potentially fatal complication. Some patients with PAS have no symptoms, while others may have fever, chills, abdominal pain, diarrhea, and worsening ascites.

Bleeding esophageal varices

In cirrhosis of the liver, scar tissue blocks the flow of blood to the heart of the intestines and increases the pressure in the portal vein (portal hypertension). When the pressure in the portal vein is large enough, which causes blood flow to the liver through the veins with less pressure to reach the heart. The most common veins through which blood passes through the liver are the veins along the lower esophagus and upper stomach.

Because of increased blood flow and thereby increasing the pressure, the veins of the lower esophagus, upper stomach and the expansion and then called esophageal and gastric varices, portal pressure, varicose veins more and more likely that a patient is bleeding from varices in the esophagus or stomach.

Bleeding varices are often severe and, without immediate treatment can be fatal. Symptoms of bleeding varices are vomiting blood (vomiting can be red blood mixed with clots or “coffee” in appearance, the latter due to the effect of acid in the blood), passage of stool that is black and tarry stools, due to changes in the blood that passes through the intestine mane () and dizziness or fainting hypotension (caused by a fall in blood pressure, especially when standing in the supine position).

It may also be bleeding from varices that form in other parts of the intestine, for example, the colon, but this is rare. For unknown reasons, patients hospitalized with active bleeding from esophageal varices are at high risk for spontaneous bacterial peritonitis.

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