Search Results for

Useful Vitamins for Diabetes Treatment

The classic diabetes therapy focuses mainly on drugs: anti-diabetic agents and insulin therapy. I think this is a big mistake can be the vital nutrients (especially vitamins) and trace the blood glucose levels are greatly improved.

Vitamin E in diabetes
With this vitamin, you reduce your risk of forming a blood clot. In addition, it reduces the formation of LDL-cholesterol, which can lead to atherosclerosis. Vitamin E is contained in 40 g of walnuts, 19 g of sunflower oil or 200 g of black roots – with these sets you meet your daily requirement of 12 mg of vitamin E.

Vitamin B for diabetic
The vitamins of the B-series provide the nervous system with energy. But even among diabetics, the B-vitamins are lost in large quantities in the urine. Are important in diabetes, vitamins B1 (vitamin B1 in diabetes), B6 and B12. These quantities can be found for example in 240 g of brown rice, 400 grams of bananas, 300 g 200 g avocado Brie, 100g goat’s cheese, 200 ml buttermilk 200 g chanterelles or 400 g of peas.

Folic acid in diabetes
Also, the folic acid is actually a B vitamin. It is absent, this leads to increased homocysteine levels, as many as 30% of all diabetics are affected. This increases the risk of vascular diseases is rising even further, and suffer with it the risk of heart attack or a stroke. You need pg per day 160th Include this amount in 300 g spinach, 300 g of Chinese cabbage, 200 g or 200 g fennel beetroot.

Zinc in diabetes
Even the trace element zinc is increased in diabetics excreted in the urine. Lack of zinc, the glucose-impaired recording continues. Furthermore, there is disruption in wound healing, as well as a general susceptibility to infections.

You need a day 10 to 20 mg of zinc. This amount of zinc can be found in 500 g of whole wheat bread, 100 g wheat germ, 190 g of dried mushrooms or 230 g lean hard cheese. Make a note of these foods than those who are zinc-containing. You should always make time to be back on your diet. However, you can cover your zinc requirement as diabetes through diet alone. I tell my patients with diabetes also eat a respiratory tract infections.

Magnesium in diabetes
Magnesium is needed for all processes, which extract energy. Laboratory values, however, show that half of diabetics have too much low magnesium levels. It is this deficiency can be dangerous. Too low magnesium levels reduces the effect of insulin. Too little magnesium is also partly responsible for damage to the retina. You need 300 to 600 mg per day of magnesium.

Include this amount in 200 g of spinach, 3 bananas, 100 g of sesame seeds, or 100 g of sunflower seeds. Unfortunately, the determination of magnesium in the blood no cash value amount and is therefore not intended for the vast majority of patients.

Taurine in diabetes
Taurine is an amino acid that you take on protein products themselves. Taurine reduces the clumping of platelets, and thereby protects against the formation of blood clots – and thus as a prior heart attack and stroke, diabetes. It is therefore possible to feed with high-quality protein in order to provide themselves with adequate taurine to. Suitable protein sources are common, for example, potatoes with cottage cheese or lentils, legumes.

Incoming Search Terms :

Diabetes Insulin Therapy and Management

If a therapy with Anti-diabetics is not sufficient with you, you should need a diabetes insulin therapy. That is the case when the insulin-producing cells are exhausted and have stopped production. In this case you get “normally” the so-called “conservative given insulin therapy” prescribed.

In this diabetes treatment (diabetes insulin therapy) you receive certain times when you need to inject a precise amount of insulin: this is usually two or three times per day. You must meet these requirements exactly. Accordingly, you are not very flexible when it comes to postpone meals. Your meals are not exactly distributed according to plan, it can easily lead to hypoglycaemia.

But there are other ways of developing diabetes insulin therapy:

- Diabetes intensified insulin therapy

The intensive insulin therapy is also known as basal-bolus therapy. This form of therapy is performed today in most type 1 diabetics. In the basal-bolus therapy is long-acting insulin and short-acting insulin is used. With two to three injections, with which you inject insulin, a delay, cover the basic needs of your body to insulin. In addition, you get a “normal” acting insulin, with which you meet the needs at mealtime (bolus).

Since the basic need is met, you can plan your meals free and not tied to such a strict timetable. Still need between four and eight injections a day. The resort used insulin types is possible, however, on small Injiziergeräte (such as pens). This is for most people, however, relatively easy. The intensive insulin therapy demonstrated in studies that may be replaced by this (and with the subcutaneous insulin infusion), the late complications of diabetes occur best be prevented.

- Diabetes supplemental insulin therapy

The supplemental (substitute) insulin therapy (SIT) is applied in a diabetic whose pancreas is not itself able to provide the basic care of the body to insulin (Type-2) diabetics. As the term “supplemental” implies, is replaced, only the amount of insulin that is necessary for normal human metabolism.

Usually, the patient injects himself with meals called insulin analogs, which have compared to human insulin, a faster and faster effectiveness. In this way, the blood sugar spikes that are caused by ingestion of food, is prevented. Because of the rapid effect of insulin must be complied with no time lag between injection and meal. This type of diabetes therapy provides people with diabetes also the possibility to freely design his meals, since the insulin dosage is adjusted to the eating carbs and not vice versa.

- Diabetes continuous subcutaneous insulin infusion

For the continuous subcutaneous insulin infusion, you will not have to worry about your insulin injection. They carry a small pump on the body, which is backed by the insulin-basic (Basic). For the bolus, the demand for meals, you simply press a small button on the device.

These pumps are suitable if your metabolism is still healthy. However, you must be very well trained in the use of such a pump and well informed about diabetes and to know the relationship decision. At the same time you need to estimate a degree of “body awareness” in order to possibly symptoms can occur.

Conclusion : There are several ways of insulin therapy, which can come to you when your life changes are used. So it is worthwhile for you to be well informed, because you can discuss with your doctor the best treatment of diabetes.

Incoming Search Terms :

The Most Frequent Cirrhosis Causes

There are some causes of cirrhosis, such as alcohol, non-alcoholic fatty liver disease (NAFLD), cryptogenic cirrhosis, chronic viral hepatitis, Inherited disorders, primary biliary cirrhosis, primary sclerosing cholangitis, autoimmune hepatitis, biliary atresia, and cardiac chronic heart failure as cirrhosis causes problems.

• Alcohol cause hepatic cirrhosis

Alcohol is a very common cause of cirrhosis, especially in the western world. The development of cirrhosis depends on the amount and regularity of alcohol consumption. Chronic high levels of alcohol consumption for liver cell damage. Thirty percent of people drinking daily for at least eight to sixteen ounces of hard liquor or the equivalent of fifteen or more years will develop cirrhosis. Alcohol causes a range of diseases of the liver to be simple and uncomplicated fatty liver (steatosis), to more severe fatty liver with inflammation (nonalcoholic steatohepatitis or hepatitis), cirrhosis.

• Non-alcoholic Fatty Liver Disease (NAFLD)

NAFLD, better known as Non-Alcoholic Fatty Liver Disease refers to a broad spectrum of liver diseases such as alcoholic liver disease, ranging from simple steatosis to non-alcoholic steato-hepatitis (NASH) to cirrhosis. All stages of NAFLD have in common the accumulation of fat in liver cells. The term is used because NAFLD nonalcoholic occurs in people who do not consume excessive amounts of alcohol, however, in many respects, the microscopic image of non-alcoholic fatty liver is similar to what can be seen in liver disease caused by excessive alcohol consumption. Is NAFLD associated with a condition called insulin resistance, which in turn is associated with metabolic syndrome and type 2 diabetes mellitus. Obesity is a major cause of insulin resistance, metabolic syndrome, type 2 diabetes. NAFLD is the most common liver disease in the United States and is responsible for 24% of all liver diseases. In fact, the number of livers that are transplanted from non-alcoholic fatty liver, cirrhosis is related to the increase. The public health administrators that the current epidemic of obesity dramatically increases the development of nonalcoholic fatty liver and cirrhosis in the population.

• Cryptogenic cirrhosis (cirrhosis due to unidentified causes)

Cirrhosis due to unidentified causes, better known as Cryptogenic-Cirrhosis is a common reason for liver transplantation. It’s called cryptogenic cirrhosis because for years doctors have been unable to explain why a proportion of patients with cirrhosis developed. Doctors now believe that cryptogenic cirrhosis due to NASH (nonalcoholic steatohepatitis) caused by long-standing obesity, type 2 diabetes and insulin resistance. The fat in the liver of patients with NASH is estimated to disappear with the onset of cirrhosis, and this makes it difficult for physicians to establish the link between NASH and cryptogenic cirrhosis long. An important clue leading to NASH cryptogenic cirrhosis is the discovery of a large number of Nash in the new liver of patients undergoing liver transplantation in cryptogenic cirrhosis. Finally, a French study suggests that patients with NASH have a similar risk of developing cirrhosis patients with long-term infection with hepatitis C. (See below). However, it is expected that progression to cirrhosis from NASH to be slow and the diagnosis of cirrhosis is usually performed in patients in the sixties.

• Chronic Viral Hepatitis

Viral hepatitis chronic is a condition where chronic hepatitis B or chronic hepatitis C affects the liver for years. Most patients with viral hepatitis develop chronic hepatitis and cirrhosis. For example, most patients infected with hepatitis A recover completely within a few weeks without developing chronic infection. However, some patients infected with hepatitis B and most patients infected with hepatitis C develop chronic hepatitis, which in turn leads to progressive liver disease leading to cirrhosis and sometimes liver cancer.

• Inherited (genetic) disorders

Inherited genetic disorders that cause the accumulation of toxic substances in the liver, leading to tissue damage and cirrhosis. Examples include the abnormal accumulation of iron (hemochromatosis) or copper (Wilson’s disease). In hemochromatosis, patients inherit a tendency to absorb too much iron from food. Over time, iron accumulation in various organs in the body causes cirrhosis, arthritis, heart muscle damage leading to heart failure, and testicular dysfunction causing loss of sexual appetite. Treatment aims to prevent organ damage from the removal of iron in the body by bleeding (blood removal). In Wilson’s disease, is an inherited abnormality in one of the proteins that control copper in the body. Over time, copper accumulates in the liver, eyes and brain. Cirrhosis, tremor, psychiatric disorders and other neurological disorders occur if the condition is not treated quickly. Treatment with oral medication that increases the amount of copper from the body in urine.

• Primary biliary cirrhosis (PBC)

PBC, better known as Primary Biliary Cirrhosis is a liver disease caused by abnormal immune system that is predominantly female. Abnormal immunity in PBC causes chronic inflammation and destruction of small bile ducts in the liver. The bile ducts are the passages in the liver, bile travels to the intestine. Bile is a liquid produced by the liver that contains substances necessary for digestion and absorption of fat in the intestine, and other compounds that are waste products such as bilirubin, a pigment. (Bilirubin is produced by the breakdown of hemoglobin in red blood cells.). Along with the gallbladder, bile ducts form the biliary tract. In the PBC, the destruction of small bile duct blocks the normal flow of bile into the intestine. As the inflammation continues to destroy more of the bile ducts, also extends to destroy liver cells nearby. That the destruction of hepatocytes of products, the scar tissue (fibrosis) forms and propagates in the areas of destruction. The combined effects of ongoing inflammation, scarring, and the toxic effects of waste accumulation leads to cirrhosis.

• Primary sclerosing cholangitis (PSC)

PSC, better known as Primary Sclerosing Cholangitis is a rare disease that occurs frequently in patients with ulcerative colitis. In the PSC, the large bile ducts outside the liver become inflamed, narrowed and blocked. Outflow obstruction of the bile duct in biliary tract infections and jaundice, and eventually causes cirrhosis. In some patients, bile duct injury (usually after surgery) can also cause obstruction and cirrhosis of the liver.

• Autoimmune hepatitis

Autoimmune hepatitis is a liver disease caused by abnormal immune system that occurs most often in women. The abnormal immune activity in autoimmune hepatitis resulting in inflammation and progressive destruction of liver cells (hepatocytes), which eventually leads to cirrhosis.

• Babies can be born without bile ducts (biliary atresia)

Biliary atresia, also known as infant can be born without bile ducts and eventually develop cirrhosis. Other babies are born without enzyme vital for control of sugar that leads to the accumulation of sugars and cirrhosis. On rare occasions, the absence of a specific enzyme can cause cirrhosis and scarring of the lungs (alpha-1 antitrypsin deficiency).

• Other less common causes of cirrhosis include unusual reactions to certain drugs and exposure to toxins, and chronic heart failure (cardiac cirrhosis)

In some parts of the world (especially North Africa), infection of the liver parasites (schistosomiasis) is the most common cause of liver disease and cirrhosis.

Incoming Search Terms :

MODY Diabetes, Maturity-Onset Diabetes of the Young

MODY (Maturity Onset Diabetes of the Young ) is an autosomal dominant inherited form of diabetes. MODY is a mild form of diabetes resembling type 2 diabetes mellitus. In diabetes MODY does the pancreas (pancreatic) in an insufficient increase in blood glucose (blood sugar) and therefore gives too little insulin. Insulin is a hormone that ensures that glucose (sugar) is absorbed into the body cells. Glucose serves as fuel for the cells. There are six known diabetes MODY type, which is diverse in form and severity.

Characteristics of MODY Diabetes are:
- It usually starts at a young age.
- Few years MODY can be treated without insulin and therefore insulin independent.
-There are at least four MODY as first-degree relatives (parents, children, brothers, sisters) at a young age, diabetes arose, at least for two to five years could be treated without insulin. At least two persons is the disease diagnosed for the 25th year of life.

Diabetes MODY Diagnosis
MODY is suspected on the above characteristics. Moreover, the diagnosis may be confirmed by genetic research.

MODY Diabetes Treatment
Treatment of MODY diabetes varies by type. Most mensem with MODY-3 respond well to low dose sulphonylureas, which are drugs that stimulate the production of insulin. MODY-2 at its dietary advice often enough. Other types of MODY are usually treated with insulin.

According to medical research, it is estimated that 2 to 4% of all people with diabetes mellitus type 2, MODY does. Diabetes MODY-3 in United States and Europe (France, Italy, Poland, Russia, Spain, United Kingdom)  is the most common, followed by diabetes MODY-2 and MODY-1 diabetes.

Incoming Search Terms :

Cause of Diabetes Type 2 and Treatment

The cause of Type-2 Diabetes Mellitus (DM Type-2) is the innate or acquired immunity to insulin (insulin resistance). Such insulin resistance may be present the result of a long-term food intake, which frequently leads to elevated blood glucose levels. The high concentration of glucose in the blood it can also increase insulin levels. In response, the number of insulin receptors decreases the body’s cells and they are less sensitive.

Thus, although actually very much insulin is secreted by the pancreas that extends from the crowd but not to lower the blood sugar levels. It develops a so-called relative insulin deficiency.

To keep blood sugar levels still low, the pancreas is working flat out to produce more insulin. The islet cells are thereby burdened so much that they exhaust for years and finally perish. As a result, developed diabetes mellitus. In United States, Europe, Australia and other region in Asia especially China and India, this form of diabetes due to poor eating habits has become particularly common.

More than 80 percent of Diabetes Type 2 are overweight and obesity is the crucial mellitus as a risk factor for the development of diabetes. In the majority of sufferers, a number of typical changes are found, which are grouped under the term Metabolic Syndrome or “wealth syndrome.” These include an abdominal type obesity, lipid disorders (high cholesterol), high blood pressure and sugar metabolism disorder first (insulin resistance), Type 2 Diabetes Mellitus later.

The hereditary predisposition plays a role in the development of diabetes mellitus type 2 In children, a parent with Type-2 Diabetes Mellitus (Diabetes Type 2), the probability of a subsequent Type-2 Diabetes Mellitus would enter up to 50 percent. The risk for identical twins is 100 percent.

Incoming Search Terms :